mirabilis clone with chromosomal AmpC-type beta-lactamase was reported in the literature ( 16). Proteus mirabilis may become resistant to β-lactams upon the acquisition of heterologous β-lactamase genes ( 15). Proteus mirabilis is the third most common cause of nosocomial infections accounting for 90% of all Proteus infections ( 11).Ī major problem in wound infections is the ever-rising antimicrobial resistance in P. It is mostly found in natural environments and responsible for the infection of the pulmonary system, burns, skin, eyes, ears, nose, and the urinary tract, as well as gastroenteritis ( 9, 10). Proteus mirabilis is a Gram-negative, rod-shaped, facultative, anaerobic, non-capsulated, non-spore forming, and motile bacterium ( 8). There are several species of Proteus including Proteus mirabilis, P. Proteus species belong to the Enterobacteriaceae family ( 1). Wound Infection Antibiotic PCR Proteus mirabilis 1. The PCR technique is one of the sensitive, timesaving, specific, and cost-effective ways for the identification of the pathogenic genes of P. mirabilis and can help prevent the spread of infection and reduce the cost of treatment. The use of aminoglycosides such as gentamycin and amikacin is effective against P. mirabilis in wound infection and its antimicrobial sensitivity are major problems worldwide. mirabilis showed clear bands of 533 bp of the ureC1 gene. mirabilis than burn wounds (n = 16 3.30%).

Diabetic (n = 24 5%) and surgical (n = 24 5%) wounds were more affected by P.

mirabilis was the highest in 16 - 30-year-old group (n = 32 6.70%), followed by the age groups of 5 - 15 (n = 24 5%) and 30 - 50 years (n = 8 1.60%). The age distribution showed that the infection of wounds with P. mirabilis were more common in male patients (n = 40 8.3%) than in female patients (n = 24, 5%).

The results showed that wounds infected with P. There were 64 (13.3%) samples positive and 416 (86.6%) samples negative for P.

Prodeus infection